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Homework answers / question archive / What is true of the rate of cancer in post cancer treatment patients? What are the classes of antimetabolite anticancer agents? and what do they target? How do antifolates function? How do nucleosides function and what is the prototype? What is the target of bloth cell cycle dependent and indpendent drugs? What is the general makeup of DNA? What part of DNA do antimetabolites mimic? What is the difference between RNA and DNA? What is the target of antibiotics? When do alkylating agents bind to DNA? What is a 5-Fluorouracil?
Anti-cancer treatments can often cause other cancers later on due to effecting the DNA
Antimetabolites target the S-phase (DNA synthesis) and are cycle specific -- antifolates and nucelosides
block folate from incorporating into DNA
interfere with the synthesizing of new DNA -- 5-fluorouracil
DNA
deoxyribose (sugar back bone) and A, G, T, C -- ribonucleic base pairs -- they are similar to these components needed for DNA synthesis
RNA -- ribonucleic backbone acid and instead of T's you have U's
antibiotics disrupt DNA function
in both cycling and non cycling cells
5-FU -- anti-metabolite is most effective against tymors with high growth rate and works as a false metabolite to prevent DNA synthesis
