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University of Illinois, Chicago NURS 531 Chapter 38: Amino acid neurotransmitters MULTIPLE CHOICE 1)The N-methyl-D-aspartate receptor for excitatory amino acids is positively modulated by g-amino butyric acid tyrosine glycine phenylalanine alanine 2
University of Illinois, Chicago
NURS 531
Chapter 38: Amino acid neurotransmitters MULTIPLE CHOICE
1)The N-methyl-D-aspartate receptor for excitatory amino acids is positively modulated by
- g-amino butyric acid
- tyrosine
- glycine
- phenylalanine
- alanine
2. The pathway leading to long-term potentiation of neuronal activity includes
- inhibition of nitric oxide synthase
- sustained blockade of N-methyl-D-aspartate receptors
- feedback inhibition by nitric oxide
- activation of protein kinase C by calcium
- inhibition of metabotropic receptors
3. Baclofen is a selective agonist for the
- kainite receptor for excitatory amino acids
- N-methyl-D-aspartate receptor for excitatory amino acids
- metabotropic receptor for excitatory amino acids
- g-amino butyric acid-A (GABAA) receptor
- g-amino butyric acid-B (GABAB) receptor
4. Activation of AMPA (a-amino-5-hydroxy-3-methyl-4-isoxazole propionic acid) receptors for excitatory amino acids leads to
- fast inhibitory neurotransmission
- fast excitatory neurotransmission
- modulation of postsynaptic excitatory neurotransmission
- slow excitatory neurotransmission
- slow inhibitory neurotransmission
5. The glycine receptor blocked by strychnine is a
- ligand-gated chloride channel
- ligand-gated calcium channel
- voltage-gated calcium channel
- G-protein-coupled receptor
- protein kinase receptor
6. The binding of g-amino butyric acid (GABA) to the GABA receptor is increased by
- flumazenil
- picrotoxin
- diazepam
- baclofen
- gabapentin
7. Which substance blocks ion channels of N-methyl-D-aspartate receptors for excitatory amino acids? 2.
3.
- ketamine
- cycloserine
- kynurenic acid
- cyclothiazide
- kainite
8. Activation of metabotropic excitatory amino acid receptors leads to
- opening of chloride ion channels
- opening of cation channels
- formation of inositol triphosphate
- formation of cyclic AMP
- activation of protein kinase A
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