Homework answers / question archive / University of Illinois, Chicago NURS 531 Chapter 5: Cell proliferation, apoptosis, repair and regeneration MULTIPLE CHOICE   1)In the death receptor pathway, activation of tumor necrosis factor receptors causes the receptors to trimerize and complex an adapter protein, thereby leading to activation of   mitochondrial pathways caspase-8 pro-apoptotic factor Bcl-2 a p53 protein DNA transcription factors 2

University of Illinois, Chicago NURS 531 Chapter 5: Cell proliferation, apoptosis, repair and regeneration MULTIPLE CHOICE   1)In the death receptor pathway, activation of tumor necrosis factor receptors causes the receptors to trimerize and complex an adapter protein, thereby leading to activation of   mitochondrial pathways caspase-8 pro-apoptotic factor Bcl-2 a p53 protein DNA transcription factors 2

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6.    Matrix metalloproteinases are responsible for

 

1. inhibition of neoplastic cell growth
2. adjusting the cell cytoskeleton
3. degradation of extracellular matrix
4. initiating angiogenesis
5. activation of the cell cycle

 

 

7.    Antisense compounds active against antiapoptotic factors are potentially useful for treating

 

1. myocardial infarction
2. leukemia
3. Parkinson’s disease
4. multiple sclerosis
5. impaired wound healing

8.    Bevacizumab is a monoclonal antibody that targets

 

1. caspase-3
2. metalollproteinase
3. cyclin
4. vascular endothelial growth factor
5. promoters of apoptosis

 

 

9.    Mitochondria participate in apoptosis by releasing

 

1. apoptotic protease activating factor
2. caspase-9
3. cytochrome c
4. p53 protein
5. inhibitors of apoptosis

10.   Progress through the cell cycle is stimulated by

 

1. cyclin-dependent kinases
2. phosphorylated Rb protein
3. check point factors
4. p53 protein
5. inhibitors of kinases