University of Illinois, Chicago NURS 531 Chapter 21: The heart MULTIPLE CHOICE 1)Delayed after-depolarizations in cardiac tissue typically result from an increased influx of sodium potassium calcium chloride sodium and potassium 2. The re-excitation of cardiac tissue prior to the next cardiac impulse occurs in the process known as re-entry ectopic automaticity atrioventricular block atrial fibrillation bradycardia 3. In persons with atrial fibrillation, digoxin may slow the ventricular rate by decreasing sinoatrial node automaticity increasing ventricular contractility inhibiting the Na+/K+ pump reducing the calcium transient slowing atrioventricular conduction 4. Sotalol exerts an antiarrhythmic effect primarily by slowing ventricular conduction blocking sodium channels slowing atrioventricular conduction increasing the effective refractory period decreasing action potential duration 5. Drugs that increase serum digoxin levels include 1. furosemide 2. cholestyramine 3. amiodarone 4.

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University of Illinois, Chicago NURS 531 Chapter 21: The heart MULTIPLE CHOICE 1)Delayed after-depolarizations in cardiac tissue typically result from an increased influx of sodium potassium calcium chloride sodium and potassium 2

Nursing Jul 07, 2021

University of Illinois, Chicago

NURS 531

Chapter 21: The heart MULTIPLE CHOICE

1)Delayed after-depolarizations in cardiac tissue typically result from an increased influx of

sodium
potassium
calcium
chloride
sodium and potassium

2. The re-excitation of cardiac tissue prior to the next cardiac impulse occurs in the process known as

re-entry
ectopic automaticity
atrioventricular block
atrial fibrillation
bradycardia

3. In persons with atrial fibrillation, digoxin may slow the ventricular rate by

decreasing sinoatrial node automaticity
increasing ventricular contractility
inhibiting the Na⁺/K⁺ pump
reducing the calcium transient
slowing atrioventricular conduction

4. Sotalol exerts an antiarrhythmic effect primarily by

slowing ventricular conduction
blocking sodium channels
slowing atrioventricular conduction
increasing the effective refractory period
decreasing action potential duration

5. Drugs that increase serum digoxin levels include

1.	furosemide
2.	cholestyramine
3.	amiodarone
4.	enalapril
5.	hydrochlorothiazide

6.	Verapamil is indicated for the treatment of
1.	supraventricular tachycardia
2.	ventricular tachycardia
3.	atrial fibrillation
4.	ventricular fibrillation
5.	cardiac asystole

7.	In persons who have had a myocardial infarction, flecainide has been shown to
1.	decrease the incidence of ventricular fibrillation
2.	decrease the incidence of atrial fibrillation
3.	decrease the incidence of sudden death
4.	increase the frequency of ventricular ectopic beats
5.	increase the incidence of sudden death
	Answer e: increase the incidence of sudden death
8.	The pharmacokinetic attributes of amiodarone include
1.	rapidly metabolized and excreted
2.	a very short elimination half-life
3.	a very long elimination half-life
4.	distributed only to extracellular fluid
5.	limited binding to plasma proteins

9.	Dobutamine is useful in treating acute heart failure because of its ability to
1.	increase heart rate more than contractile force
2.	increase contractile force more than heart rate
3.	produce a negative dromotropic effect
4.	cause cardiac stimulation and vasoconstriction
5.	inhibit phosphodiesterase

10.	Digoxin slows AV conduction and decreases heart rate by
1.	activating cardiac muscarinic receptors
2.	blocking cardiac adrenergic beta-1 receptors
3.	antagonizing cardiac calcium channels
4.	increasing vagal outflow from the CNS
5.	increasing cardiac contractile force

11.	The half-life of digoxin is considerably longer in persons with
1.	renal insufficiency
2.	pulmonary edema
3.	atrial fibrillation
4.	hypertension
5.	endocarditis

12.	Which drug has been shown to reduce mortality in persons who have had a myocardial infarction?
1.	amiodarone
2.	lidocaine
3.	propranolol
4.	amiodarone
5.	verapamil

13.	Hypothyroidism is most likely to occur in persons who are taking
1.	sotalol
2.	flecainide
3.	timolol
4.	amiodarone
5.	adenosine

14.	Nitroglycerin relieves typical angina pectoris in part by
1.	increasing cardiac stroke volume
2.	dilating coronary arterioles
3.	decreasing cardiac oxygen consumption
4.	constricting collateral vessels in the heart
5.	increasing blood pressure

15.	Nitroglycerin and other organic nitrates produce their vasodilator effect by
1.	increasing formation of cyclic AMP
2.	increasing formation of cyclic GMP
3.	blocking calcium channels
4.	blocking adrenergic alpha receptors
5.	activating potassium channels

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